IntegriGen Master Brand
ENGINEERING DERMAL INTEGRITY

Next-Generation Living Cell Therapies for DEB

The team that pioneered first-generation fibroblast gene therapy has come together to build what comes next: durable, non-viral autologous and allogeneic cell medicines engineered to restore functional Collagen VII and durably treat Dystrophic Epidermolysis Bullosa (DEB) wounds, addressing both recessive (RDEB) and dominant (DDEB) forms.

Payload Size
10 kb+
Full COL7A1 + Introns & Controllers
Therapy Modality
Non-Viral
Autologous & Allogeneic Cell Medicines
Engine Platform
RAPTOR OS™
BSD Integrated Technology System
Target Tissue Architecture
COL7A1 Restoration
Epidermal Barrier Stratum corneum & keratinocyte protective envelope
Surface
Dermal-Epidermal Junction (DEJ) DEB Blistering Fault Line

Deficiency in functional Type VII Collagen causes blistering and structural detachment upon minor friction.

Dermal Compartment Cellular Niche
IntegriGen Engineered Fibroblasts: Injected locally to engraft within the dermis and continuously synthesize full-length Collagen VII homotrimers.
Direct local delivery restores anchoring fibrils to durably stabilize skin layers. Durable MoA
01 / Biological Rationale

The Structural Biology of DEB (RDEB & DDEB)

Dystrophic Epidermolysis Bullosa (DEB) is caused by genetic disruption across the exons, introns, and transcriptional control regions of the COL7A1 gene. In recessive DEB (RDEB), functional Type VII Collagen is absent. In dominant DEB (DDEB), mutant chains exert a dominant-negative effect that disrupts anchoring fibril assembly. IntegriGen addresses both with durable, non-viral living cell therapies.

01

Anchoring Fibril Loss & Genetic Disruption

Mutations across the 118 exons, introns, and regulatory control regions of COL7A1 eliminate functional Type VII Collagen. IntegriGen’s engineering strategy restores full-length COL7A1 expression with intact transcriptional regulation to re-establish dermal-epidermal attachment.

Field Status: DEB (RDEB & DDEB) Root Cause
02

Current Modality Limits

Topical viral-vector approaches have demonstrated clinical efficacy, but treatment may require repeated wound-specific application. Surgical cell-sheet approaches require specialized procedural logistics. These approaches do not necessarily provide a durable, resident cellular source of Collagen VII within the dermis.

Field Status: Validated Market
03

Continuous Dermal Production

By harvesting patient fibroblasts, non-virally integrating full-length COL7A1 with optimized transcription controllers, and delivering them locally, IntegriGen creates living dermal "factories" capable of sustained therapeutic Collagen VII production.

Lead Program: NTGN-001 (GM-Fibroblasts)
FIG 1.0 · Cellular Mechanism of Action

Normal vs. RDEB vs. NTGN-001 Genetically Modified Fibroblasts

©DTBP-DTWB 2026 · DocTom BioProductions
Cellular Mechanism of Action: Normal Fibroblasts, RDEB Fibroblasts, NTGN-001 GM-Fibroblasts (DocTom BioProductions ©DTBP-DTWB 2026)
Normal Fibroblasts

Healthy dermal fibroblasts synthesize and assemble Type VII Collagen homotrimers, forming dense looping anchoring fibrils that secure the epidermal basement membrane to the dermis.

RDEB Fibroblasts

Mutant fibroblasts fail to produce functional Type VII Collagen. Complete absence of anchoring fibrils causes severe blistering and mechanical skin detachment at the DEJ fault line.

NTGN-001 GM-Fibroblasts

Administered genetically modified fibroblasts continuously express and secrete full-length Type VII Collagen in situ, rebuilding anchoring fibrils and restoring mechanical dermal integrity.

02 / Integrated Technology Operating System

RAPTOR OS

BioSolution Designs’ Integrated Technologies

Raptor OS is BioSolution Designs’ integrated technology operating system for the development of paradigm shifting advanced therapies that are large (>10kb), complex, and / or encode multiple active therapeutic effectors (multigenic). It brings together five coordinated technology pillars—Gene Delivery, Genome Context, Gene Expression, Effector Engineering, and BioManufacturing—so therapeutic programs can be designed, controlled, and translated within a single modular architecture.

Pillar 01 Gene Delivery*
Non-Viral Delivery LAVNAV*
Large Cargo Viral MEGA*
RNA & Protein Delivery ENGINEERED EXOSOMES*
High-Capacity Vectorization
Pillar 02 Genome Context
Genome Integration INTEGRASE
Genome Modification BoP NUCLEASE
Directed Positioning MATS™
Site-Specific Insertion
Pillar 03 Gene Expression
Transcription Controller
TRANSCRIPTION UNIT

Embedded Promoters, Introns, UTRs & Enhancer Elements

Inducible Systems SWITCH* Rheostatic Gene Tuning
Tunable Expression Control
Pillar 04 Effector Engineering
Disease-Specific Effectors NOVEL
Full-Length Proteins OPTIMIZED (COL7A1)
Structural Integrity HOMOTRIMER ASSEMBLY
Large Cargo Structural Design
Pillar 05 BioManufacturing
ccDNA Vector Synthesis BoP MULTIGENE MFG
Affinity Capture & Beads RAPTOR BINDER BEADS
Host Cell Production IMMORTAL CELLS
Plasmid-Free Scalable Footprint
Foundation Layer Enabled by Bird of Prey (BoP®) Complex DNA Design and Assembly Technology
BioSolution Designs · Boulder, CO
* Technology component of BioSolution Designs’ broader Raptor OS™ engine; not currently licensed by IntegriGen Therapeutics.
03 / Pipeline & Assets

Therapeutic Asset Portfolio

Advancing lead autologous candidate NTGN-001 through discovery and translational characterization.

NTGN-001 LEAD AUTOLOGOUS ASSET

Autologous Fibroblast Therapy for DEB (RDEB & DDEB)

Targeting durable COL7A1 restoration in chronic blistering wounds

Non-viral ccDNA + Raptor OS™ Enrichment
Discovery Discovery / In Vitro & In Vivo Characterization Discovery Stage
NTGN-002 ALLOGENEIC / OFF-THE-SHELF

Allogeneic MSCs for Accelerated Tissue Repair

Off-the-shelf stromal cell therapies for severe chronic wound beds with enhanced durable effect

Engineered Allogeneic MSCs
Discovery / Early Preclinical Discovery Stage
04 / Leadership & Governance

Decades of Proven Fibroblast & Gene Therapy Precedent

IntegriGen unites key inventors, clinical manufacturing authorities, and executives who developed first-generation fibroblast therapies through the historic Fibrocell–Intrexon collaboration.

Leadership & Governance
Dr. Thomas D. Reed, PhD

Dr. Thomas D. Reed, PhD

Founder, CEO & Chief Scientific Officer CEO / CSO · Founder

Founder & CEO of BioSolution Designs. 20+ year tenure at Intrexon (Precigen) inventing foundational platforms (UltraVector®, RheoSwitch®). Led the 2017 GenVec acquisition and AdenoVerse® platform, inventing the FDA-approved PAPZIMEOS (Aug 2025).

John Maslowski

John Maslowski

Board of Directors Governance & Board

Former CEO of Fibrocell Science / Castle Creek leading the FCX-007 (D-Fi) RDEB Phase 1/2 trial (NCT02810951; 80% wound healing at 12 wks). Currently President & CEO of Forge Biologics and Chair of the Board of Directors for DEBRA of America.

Product Development & Manufacturing
Erin Collins

Erin Collins

Head of Product Development & Regulatory Product Development & Regulatory

Molecular biologist focused on design and optimization of biologics, with a focus on transition from laboratory into clinical trial initiation.

Wil Thompson

Wil Thompson

Head of CMC & Manufacturing Product Development & Manufacturing

Oversees autologous cell processing, cGMP facility operations, analytical release testing, and clinical technology transfer protocols.

Product Design
Dr. Stephen Schauer, PhD

Dr. Stephen Schauer, PhD

Head of Synthetic Biology & Regulation Product Design & Synthetic Biology

Synthetic biologist and systems modeler focused on gene regulation circuitry and construct optimization for large multigenic payloads.

COMMS & IR
Michael Linsner

Michael Linsner

Head of Communications & Investor Relations

Comms & Investor Relations

Leads corporate communications, investor relations, and capital strategy with extensive startup capitalization experience across deep tech and life sciences.

05 / Mission & Corporate Charter
IntegriGen Therapeutics · Core Product Mission
IntegriGen Brand Mark

Dedicated to developing Advanced Therapy Product Candidates for Epidermolysis Bullosa — focusing on durable therapeutic effects while mitigating the chronic symptoms of pain and itch.

"Patients are our focus. Caregivers are essential partners. These directives allow us to create value for our stakeholders. We succeed together."

BioSolution Designs Studio Philosophy · Supporting Directive

01 · Durability Over Dosing

Direct Relief from Daily Care

We measure success not by bandage maintenance, but by genuine dermal engraftment designed to promote durable wound closure and reduce the trauma of daily dressing changes.

02 · Preclinical Rigor

Uncompromised Quality Gates

Every cell batch must satisfy strict Raptor OS™ enrichment thresholds before release. We prioritize product potency, genomic safety, and reproducibility over rapid clinical shortcuts.

03 · Global Access

Scalable Autologous Delivery

Advanced cellular therapies must not be restricted to top-tier research hospitals. We design processes for scalable, localized delivery that reaches patients globally.

Investor Relations & Diligence Inquiries

Corporate & Investor Inquiries

IntegriGen is actively engaging with institutional funds, accredited investors, and strategic partners to advance our next-generation living cell therapy pipeline for Dystrophic Epidermolysis Bullosa (DEB).

Seed Financing & Preclinical Pipeline Dedicated capital supporting lead autologous candidate NTGN-001 through translational validation and advancing our allogeneic platform.
Executive Diligence & Orbit Portal Access Direct channel to connect with executive leadership, review scientific diligence materials, and receive access credentials for the Orbit investment portal.
Corporate Headquarters
IntegriGen Therapeutics, LLC
2900 Center Green Ct, Suite 100
Boulder, CO 80301
Invention Studio & IP Platform
BioSolution Designs
Boulder, Colorado

Corporate & Investor Inquiry

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